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There has been an upward trend in occurrence of CCB-influenced gingival enlargementÄue to the unbridled use of CCBs. The surgical technique employed to treat the pathosis plays an important role in long term success of therapy. Conclusion: Dental plaque, genetic predisposition, and type of drug used are critical in the development of drug-influenced gingival enlargement (DIGE). Study selection: Clinical studies, systematic reviews, in vitro studies, and case reports. The application of various treatment options is also discussed. The article reviews contemporary and classic literature with a focus on pathophysiology, clinical presentation, and risk factors associated with this condition. Data and Sources: MEDLINE/PubMed, Scopus, ScienceDirect, and Google Search databases. The aim of the article is to discuss the development of calcium channel blocker (CCB) influenced gingival enlargement.
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The purpose of the present article is to review case reports and case series published in the last two decades and to assimilate and compile the information for clinical applications such as diagnosis and therapeutic management of DIGO. Cessation or change of drug and meticulous plaque control often leads to regression of the lesion, which however might need surgical correction for optimal maintenance of gingival health. Histopathological examination is often neglected however, it is an important aid that helps in differential diagnosis. Careful clinical examination and thorough history taking form the basis for diagnosis of DIGO. Drug variables, plaque-induced inflammation, and genetic factors are the three important factors in the expression of gingival changes after systemic medication use.
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Calcium channel blockers, anticonvulsants, and immunosuppressants are frequently implicated drugs in the etiology of DIGO.
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Drug-induced gingival overgrowth (DIGO) is a well-recognized adverse effect of certain systemic medications.